Abrasion of hard dental tissues. Tooth wear – pathological and physiological

Increased tooth wear is a pathology that requires urgent treatment. Every year this disease becomes “younger”, affecting people under 30 years of age. Intensive loss of hard tissue leads not only to aesthetic problems, but also to functional disorders of the dentofacial apparatus. Why does the disease develop, what treatment methods does it offer? modern dentistry?

The difference between natural and pathological tooth wear

Throughout life, a person's enamel gradually wears away - this is a normal process. It wears off very slowly even in children - this is how the teeth adapt to the chewing load. Normally, the thickness of the enamel decreases only in the area of ​​​​tooth contact, while the dentin is not affected. Normal is a gradual loss of the hard layers of the tooth by 0.034–0.042 mm per year.

By the age of 30, a person’s front teeth wear down slightly, and the masticatory cusps acquire a smoothed outline. By the age of 50, the enamel on contact surfaces disappears almost completely without damaging other tissues. In older people, dentin begins to wear down. If the described process accelerates, this indicates pathological abrasion of teeth.

Pathology is indicated by a decrease in the thickness of the hard layers of the elements of the dentition in young people - usually the abrasion process begins at 25–30 years of age. In humans, the height of the crown slowly decreases, its shape changes, the bite is disturbed, and the sensitivity of the units increases.

This condition can occur suddenly. Research suggests that 12% of the planet's population is susceptible to this pathological process, and in more than 60% of cases men suffer from the disease.

Classification of pathology

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There is a classification of the process of tooth abrasion, compiled depending on the type and complexity of the disease. There are 4 degrees of abrasion:


  • 1 – reduction in the thickness of the top layer of enamel;
  • 2 – complete erasure of the hard layer of the unit down to the dentin;
  • 3 – the crown is reduced by more than half, a dental cavity becomes noticeable;
  • 4 – the unit is erased to the ground.

Depending on the complexity of the disease, there are:

  • local abrasion - only one area of ​​the dentition is affected by pathology;
  • generalized - the process spreads to both jaws, but the degree of damage to units may vary.

There is also a classification that determines the plane under which the teeth were abraded:

  • horizontal – the height of a person’s crowns decreases almost uniformly;
  • vertical - the front surface of the lower and rear end upper canines and incisors (occurs in malocclusion);
  • mixed - teeth are destroyed in both planes.

Tooth wear occurs in various forms, and the severity of each may vary. However, if dentin is affected and the nerve dies, the pathological process is irreversible.

Using the classification, the doctor determines the percentage of enamel loss and the rate at which the disease progresses.

Causes and symptoms of increased abrasion

To understand why a patient develops pathology, the dentist must ask him about his lifestyle and find out about diseases in the family. Most dangerous reasons Increased tooth wear is caused by hereditary factors:

  • Congenital disorder of hard tissue formation. The disease develops due to a lack of microelements in the mother’s body during pregnancy for the development of the fetus, as well as their deficiency in the first year of the baby’s life.
  • Marble disease, osteogenesis and other ailments that are inherited.
  • Diseases associated with dysfunction thyroid gland and problems with the body's absorption of calcium.

Also, increased tooth wear is provoked by other reasons:

  • broken bite;
  • night grinding of teeth (bruxism);
  • loss of several teeth;
  • frequent intoxication of the body due to regular consumption of alcohol and smoking;
  • incorrectly performed prosthetics or unsuccessfully installed filling;
  • softening of enamel in some diseases;
  • frequent consumption of foods containing acid (juices, candies, etc.);
  • unhealthy diet, including constant consumption of sweet, starchy and hard foods;
  • bad habits - chewing the ends of pens, toothpicks and other objects;
  • taking certain medications that lead to the destruction of the hard layers of the tooth;
  • work associated with exposure to hazardous work.

With pathological abrasion in humans, the sensitivity of the enamel to temperature changes increases. Associated symptoms diseases:

  • sharp, severe pain, often appearing at night;
  • increase in interdental spaces;
  • presence of caries;
  • reducing the height of the crowns;
  • trauma to the mucous membrane due to the formation of chips and sharp edges of the teeth;
  • change in bite;
  • frequent cheek biting;
  • feeling of roughness of teeth;
  • feeling of jaws sticking together when closing them;
  • change in enamel color.

Treatment of increased tooth wear

If the patient has worn down teeth, treatment is carried out taking into account the severity of the process. The efforts of doctors are aimed at eliminating the causes of abrasion: fighting bad habits, replacing dentures, correcting bites, etc.

Pathological abrasion of teeth at an early stage is treated using remineralizing therapy - the patient is prescribed vitamin complexes, make applications with fluoride-containing preparations, and perform electrophoresis. If there are sharp edges of the teeth, they are ground down, and for bruxism, the use of a night guard is prescribed. However, most often patients consult a doctor when their teeth have already been significantly worn down. In this case, treatment is aimed at restoring the units.

Treatment of pathological wear of incisors, canines or chewing teeth is carried out using various designs. In dentistry the following are used:

  • Crowns. Metal ceramics are used to restore significantly damaged units. If a structure of increased strength is required, products made of metal or zirconium dioxide are installed. The restored tooth takes on part of the load, relieving it from its neighbors.
  • Ceramic inlays and veneers. If the wear of the front teeth is severe and has reached the dentin, the units are restored with thin plates (we recommend reading:). They are highly aesthetic and natural in appearance.
  • Stump inlays. This technique is suitable for significant tooth wear - a pin is installed in the root canal, around which a crown is built up.
  • Prosthetics with implants. When in a patient with a problem of increased abrasion, the units are destroyed to the very foundation, they are replaced with artificial material. The festered roots are removed, and a pin is installed in place of the lost element, onto which a crown is placed. The restoration procedure can take up to six months.

Treatment of pathological tooth abrasion of stages 3 and 4 necessarily begins with the restoration of the bite - installation of crowns on initial stage therapy is prohibited because they can cause malocclusion. Subsequently, the orthopedist makes and installs prostheses from the same materials (we recommend reading:). Violation of this rule may lead to the need to re-correct the bite.

If the cause of the problem is an increased load on the units, experts recommend installing durable prostheses made of metal or zirconium dioxide (see also:). Fragile ceramics, cermets or metal-plastics are not used.

Regardless of the chosen method of restoring units in case of tooth wear, doctors recommend using a mouth guard to reduce the load on the units. The design allows the muscles to get used to the new position of the teeth.

Prevention measures

To prevent abrasion and changes in the shape of teeth, you need to visit the dentist every six months - this will allow you to identify the problem in time. In addition to the preventive examination, it is necessary:

  • cure bruxism and correct bite;
  • give up bad habits;
  • restore deleted and destroyed units in a timely manner;
  • Healthy food;
  • use vitamin-mineral complexes;
  • in hazardous industries, protect teeth with special devices.

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Age-related tooth wear is a natural process associated with active use. oral cavity for crushing and chewing foods, including hard ones. Sometimes increased tooth wear is diagnosed in people under the age of 40, which indicates pathological changes in the dental tissues and the need for effective treatment.

In the article we will look at what affects the premature destruction of dental tissues, what symptoms accompany this process and how to get rid of the problem.

Tooth wear

With a normal bite, a person wears out faster outer side the lower teeth and the inner one on the upper teeth. The chewing surface of the molars regularly receives stress during food intake, which leads to the erasure of the natural cusps.

Normally, age-related erasure begins after 40 years, and the frequency of requests from men is several times higher than from women. If the problem is found in a teenager, young man under the age of 30, then we are talking about pathological abrasion.

Let's look at what causes it.

  1. Use of medications. Some aggressive drugs (for example, based on of hydrochloric acid) lead to destruction of the top layer of enamel.
  2. Heavy physical activity. Athletes and even loaders often experience pathological wear of teeth (photo above), which is associated with the tight closure of the jaws during heavy lifting.

Symptoms of pathology

Clinical picture characterized by accelerated abrasion of the top layer of the tooth (enamel) with a transition to soft fabrics(dentine).

When dentin is exposed, the tooth wears out much faster, resulting in chips, sharp edges, and gouges on the tooth.

Such defects lead to microtraumas of the mucous membrane, tongue and lips.

The initial stage is also characterized hypersensitivity enamels to temperature, chemical and mechanical factors. Sharp pain can be caused by heated or chilled food and drinks, too spicy, sour, sweet or salty dishes. Pain is also observed when touching the tooth during regular oral hygiene.

When dentin is exposed and the appearance of a replacement substance, sensitivity may temporarily decrease, while wear increases significantly.

Disease progression leads to rapid shortening of the length of the molar, which leads to visual changes in facial expressions and symmetry. Patients note drooping corners of the lips, problems and discomfort in temporomandibular joint. In some cases, hearing loss and pain in the tongue area may occur.

During this period, there is a change in the bite, which leads to inconvenience when biting and chewing food. In some cases, chewing disorders can even affect the digestive system.

The fissures are smoothed out during the erasing process. and irregularities, making the enamel surface smoother and more even. This allows you to partially get rid of early caries that lay at the bottom of such grooves.

Pathological abrasion of teeth upper jaw

In the case of increased wear of the incisors, the process sooner or later reaches the neck of the tooth, and the tooth cavity can be seen through defects in the dentin.

A deep bite is characterized by damage to the surface of the lower and upper incisors.

If the disease begins after tooth extraction, then neighboring teeth, as well as canines and incisors, are worn away.

When pathology is diagnosed in production workers chemical compounds, metal structures, as well as confectionery products, there is uniform damage to the enamel, an equally smooth surface of the teeth, and the absence of deep fissures. The surface does not have a normal glossy, but a matte shade without plaque or stone.

In some cases, exposed, smoothed dentin is noted. If the patient works in an acid production plant, the molars may be worn down to the neck. In this case, a person feels a rough tooth surface, pain, and discomfort while chewing.

On late stages The disease causes a change in the position of the teeth, their mobility and even loss. Resorption of hard tissues at dental roots and septa is also possible.

Diagnosis and treatment of the disease

Treatment of pathological tooth wear will depend on the type of pathology, initial reasons and stages of the disease.

Before starting therapy, the dentist conducts a thorough diagnosis and helps eliminate the problem that caused the increased wear. After this it is assigned rehabilitation treatment, which allows you to restore teeth and restore the aesthetics of the row.

Diagnosis involves collecting anamnesis, analyzing the patient’s subjective complaints, and determining the cause of abrasion. Dentist conducts visual inspection oral cavity and dentition, taking into account changes in bite and facial symmetry.

He also establishes the nature and degree of occlusion, notes visual symptoms, determines the hardness and resistance of tissues, and the degree of abrasion of enamel and dentin.

Dentists treat increased tooth wear

The condition of the root canals and pulp cavity must be assessed using electroodontodiagnostics, X-rays, and orthopantomography.

By using computer programs The dentist studies the model of the jaw, determines the shape, depth and degree of damage to the tooth, and the occlusal relationships of the upper and lower rows.

In the later stages, it is necessary to study the functioning of the jaw and masticatory muscles, for which X-rays, tomography of the TMJ, electromyography, etc. are used.

Treatment is usually carried out by dentists: therapist, orthopedist, orthodontist.

At the first stage, the causes of erasure are eliminated, which requires curing systemic and dental diseases, put a normal bite, change dentures or implants, give up bad habits, change diet or even place of work, restore extracted teeth in the form of crowns, etc.

At the same time, auxiliary medications are prescribed, food additives and vitamin-mineral complexes, which allow you to restore the balance of substances in the body, ensure the normal supply of calcium, mineral salts, fluorine and other microelements beneficial for teeth.

Next, hyperesthesia of the dentition is eliminated through remineralization. The patient continues to take vitamin and mineral complexes and attends physiotherapeutic procedures (electrophoresis). Applications based on fluoride-containing preparations are carried out.

Sharp edges, chips and enamel defects are ground to a smooth surface that is safe for soft tissues.

Mouth guard for bruxism

Defects and gaps in the dentition are corrected using prosthetics and implants.

To treat bruxism, customized night guards are prescribed to eliminate wear and tear on the enamel during night grinding.

The final stage is the restoration of the natural shape of the teeth (crowns, incisal edges, etc.) using filling composites, core inlays, veneers, artificial crowns, lumineers or artistic restoration.

To avoid long and expensive treatment, you need to pay timely attention to the condition of your teeth.

If you note the symptoms indicated in the article, notice a decrease in the length of the tooth or chips on the enamel surface, then make an appointment with the dentist to exclude increased tooth wear. Also, do not forget to take the microelements and vitamins necessary for dental health, purified water with normal level fluorine

Oral health is a very pressing issue for many. How beautiful and healthy a person’s teeth look can be used to judge his health, grooming and status. Ecology, stress, neglect of oral problems and non-systematic visits to the doctor contribute to the formation of various dental problems and diseases.

Pathological abrasion of teeth is a pressing problem. This is a normal physiological process of the body. In people with a correct bite, tooth enamel is scratched upper teeth begin with inside, and the lower ones, respectively, outside. The problem may arise when a person reaches a more mature age and develop into a pathological process.

According to statistical studies, 12% of the world's population is prone to pathological tooth wear (men are at greater risk - 63%). By the age of thirty, a certain layer of enamel is gradually worn away, and after fifty, the wear of the dentin layer is often recorded. If such problems begin to appear in more at a young age, we can talk about pathological nature this problem.

The main reasons for the appearance


Anatological abrasion of teeth is usually called the systematic abrasion of the enamel (in some cases, enamel and dentin) of all or several teeth. The level of neglect of this process can only be determined by a doctor using basic methods:

  1. Examination of the jaw model using a cast.
  2. Electrodiagnostics.
  3. Electromyography.
  4. Orthopantography.

Causes of dental pathology

Experts combine the main causes of this anomaly of tooth enamel into two groups, namely:

— Functional deficiency of hard dental tissues:


— Waste of human teeth due to problems associated with:

  • loss of teeth (partial);
  • bad habits, which very often become the cause of systematic injury to a person’s teeth;
  • resulting hypertonicity of the human masticatory muscles (can be formed due to tense facial muscles);
  • foodless chewing.

Classifications of increased tooth wear

The classification of this pathological disease is based on the forms and complexity of the disease.

There are main degrees of abrasion:


Taking into account the level of the erase plane, the following types are distinguished:

  • Vertical, most often found in patients with malocclusion. Only the outer side of the tooth enamel is erased.
  • Horizontal. As teeth wear down, the height of the crown decreases.
  • Mixed. When this level of the disease is reached, erasure of the two previous types is characteristic.

Depending on the complexity of the process, they differ:

  • local abrasion. In this case, one specific area is subject to erasure;
  • generalized. In this case, the process completely affects all areas of a person’s teeth.

To summarize, we can talk about numerous variants of manifestation of this dental pathology, in which all the enamel can be completely erased or only some part of it, one side - or both at once.

Symptoms of the disease

The symptoms of this disease depend both on the degree of the disease and on its course.

It's broken from the start primary view teeth. If measures are not taken, the disease develops, causing the length of the tooth to become much shorter than before. The chewing function of a person is impaired. Patients note discomfort when eating hot, cold, sweet or sour foods, which indicates the onset of hyperesthesia.


Impaired chewing function is a sign of a disease called increased tooth wear. Tooth enamel is about five times stronger than dentin, therefore, until the enamel is completely erased, the symptoms are mild, but as soon as the enamel disappears, the symptoms will become more pronounced.

This pathology requires immediate appeal See a doctor while the symptoms indicate the initial stage of the disease. If left untreated, the consequences of the disease can be deformation of the joints, changes in the lower part of the face, and the appearance of severe pain.

Diagnosis of the disease

Diagnosis of pathological abrasion of tooth enamel includes an in-depth analysis of symptoms. Because of wide range Symptoms of abrasion can only be diagnosed by a dentist, taking into account all factors and the possible presence of other pathologies.

The examination scheme includes:


  1. A complete examination and interview of the patient, studying the history of the disease to determine the forms and stage of the pathology.
  2. Inspection of external signs.
  3. Complete examination of the oral cavity and the condition of the patient’s masticatory muscles.
  4. Study of the functions of the temporal and mandibular joints.

To study the picture of the disease, radiography, tomography, and electromyography can be used.

The initial examination of the patient's face includes studying the contours of the face, its symmetry and proportionality. Specialists analyze the degree of destruction of the mucous membrane, the level of tooth wear, and the condition of hard tissues to determine possible complications during treatment.

Examination of the masticatory muscles allows one to study their condition, possible asymmetry and hypertonicity. In this case, electromyography is often used. All this helps to minimize possible complications.


Examination of the temporal and mandibular joints allows us to determine different kinds pathologies that can occur with this type of disease.

Electroodontodiagnosis, or EDI. This type diagnosis is necessary, since in the pathology of tooth abrasion, the death of the pulp very often occurs, while the patient did not observe any signs of deviation. EDI is prescribed only for the second or third degrees of the disease, since in initial stage no symptoms appear.

Diagnostics allows us to identify the main causes of increased tooth abrasion. In addition to the oral cavity, doctors focus on the condition of the temporal and mandibular joints.

Forms of treatment

Treatment of this problem takes a lot of time, this is due to a huge variety of factors influencing the problem. In addition, it is important to determine the stage of development of the disease, this will help to select correct treatment and speed up the process.


To cure pathological abrasion of the first and second degrees, doctors first stabilize the already advanced process so that the disease does not develop.

At the initial stage, doctors install temporary dentures (to begin the restoration process and maintain chewing functions). After positive dynamics are observed, temporary prostheses are replaced with permanent ones.

Treatment of more advanced stages of the disease (third and fourth) begins with restoration of the bite. At this stage, experts strictly prohibit the installation of crowns, as this may cause the patient to develop an incorrect bite. As a result of their establishment, tooth tissue may be damaged.

Making prosthetics is an important issue. At the initial levels of development of this disease, prostheses are most often made from plastic, ceramics, and sometimes the choice falls on prostheses made from precious metals. In cases where the disease has progressed, prostheses made of ceramic or metal ceramics are often used.


When installing dentures, it is important to remember that the dentures must be made of the same materials, otherwise you may end up with a reverse (re-) correction of the bite.

If the cause of pathological tooth wear is heavy load or periodic contraction of the masticatory muscles, experts recommend installing dentures that are not susceptible to cracks (more durable): made of metal-plastic or metal. In this case, metal ceramics are strictly prohibited.

Main stages of treatment:

  1. By installing temporary dentures, doctors correct the height of the bite.
  2. The adaptation of teeth to the new position is analyzed.
  3. After positive results temporary dentures are replaced with permanent ones.

Restoring the height of the bite at the first stage occurs through the installation of so-called plastic aligners.


The adaptation period is the patient's adaptation to other jaw positions. Most often, this period is characterized by severe discomfort. The patient must visit the dental office at least twice a week; this is necessary for clear monitoring and research of the results of wearing aligners by the dentist. More often average duration Wearing temporary aligners takes about two to three weeks. It is also necessary to take into account that adaptation begins from the moment the patient stops complaining about discomfort in the area of ​​the temples, mandibular joint, as well as in the area of ​​the masticatory muscles when eating food.

The third stage of treatment is the installation of permanent prostheses (final prosthetics). At this stage, special materials are selected to achieve the preservation of a correctly established bite. To achieve the best possible results, when making prostheses, doctors take into account the results obtained when wearing therapeutic mouth guards, which were installed temporarily.


The process of permanent prosthetics can take place either immediately or in stages. Aligners help determine the exact occlusal height for the patient. For the remaining areas, prostheses begin to be made after complete fixation of the first permanent prostheses.

Prevention of tooth wear

To protect yourself from the disease or from its reoccurrence, you must adhere to following rules and recommendations:


Treatment prognosis

The prognosis for treatment of this disease is generally positive. Of course, treatment takes a much shorter period of time if the patient turns to early stages diseases. In addition, younger patients are more likely to recover quickly. However, relapses of pathological tooth wear often occur, so dentists talk about the need to register patients suffering from this pathology.


Therapeutic dentistry. Textbook Evgeniy Vlasovich Borovsky

5.2.2. Abrasion of hard tooth tissues

Abrasion of tooth tissue occurs in every person, which is the result physiological function chewing. Physiological abrasion manifests itself primarily on the mounds of the chewing surface of small and large molars, as well as along the cutting edge and knobs of the fangs. In addition, the physiological surface of the teeth normally leads to the formation of a small area on the convex part of the crown at the point of contact (point contact) with the adjacent tooth.

Physiological tooth abrasion observed both in time and in permanent dentition. In the temporary dentition, when the incisors erupt, they have 3 teeth on the cutting edges, which are worn away by the age of 2–3 years.

Rice. 5.11. Abrasion of teeth.

Depending on age, the degree of physiological tooth wear increases. If up to 30 years of age, abrasion is limited to the enamel, then by the age of 40, dentin is also involved in the process, which, due to exposure, becomes pigmented in yellow. By the age of 50, the process of dentin wear intensifies, and its pigmentation takes on a brown color. By the age of 60, significant abrasion of the anterior teeth is observed, and by the age of 70 it often extends to the coronal cavity of the tooth, that is, sometimes even the contours of this cavity filled with newly formed tertiary dentin are visible on the worn surface.

Along with physiological pathological erasure, when there is an intense loss of hard tissue in one, in a group or in all teeth (Fig. 5.11).

Clinical picture. Pathological abrasion (abrasion) of hard dental tissues is quite common and is observed in 11.8% of people. Complete abrasion of the chewing cusps of large and small molars and partial abrasion of the cutting edges of the front teeth are more often observed in men (62.5%). In women, this process occurs much less frequently (22.7%). The reasons for increased abrasion may be the state of the bite, overload due to loss of teeth, improper design of dentures, household and professional harmful effects, as well as the formation of defective tissue structures.

With a straight bite, the chewing surface of the lateral teeth and the cutting edges of the front teeth are subject to wear.

As the cusps of the chewing surface wear away with age, the wear of the incisors progresses intensively. The length of the crowns of the incisors decreases and by the age of 35–40 it decreases by 1/3-1/2. In this case, instead of a cutting edge, significant areas are formed on the incisors, in the center of which dentin is visible. After dentin is exposed, its abrasion occurs more intensely than enamel, resulting in the formation of sharp edges of enamel, which often injure the mucous membrane of the cheek and lips. If treatment is not carried out, tissue abrasion progresses rapidly and the crowns of the teeth become significantly shorter. In such cases, there are signs of a decrease in the lower third of the face, which is manifested in the formation of folds at the corners of the mouth. In persons with a significant decrease in bite, changes in the temporomandibular joint may occur and, as a result, burning or pain in the oral mucosa, hearing loss and other symptoms characteristic of low bite syndrome may occur.

With further progression of the process, the abrasion of the incisors reaches up to the necks. IN in such cases through dentin The tooth cavity is visible, but its opening does not occur due to the deposition of replacement dentin.

With a deep bite, the labial surface of the lower incisors comes into contact with the palatal surface of the upper incisors and these surfaces are significantly erased.

The most pronounced tissue abrasion is observed in the absence of part of the teeth. In particular, in the absence of large molars, which normally determine the relationship of the dentition, intensive wear of the incisors and canines is observed, as they are overloaded. In addition, due to overload, tooth displacement and resorption may occur. bone tissue at the tips of the roots, interdental septa. Often, tooth wear is caused by improper design of removable and fixed dentures. When using a tooth under a clasp without an artificial crown, abrasion of the enamel and dentin at the neck often occurs. As a rule, patients complain of severe pain from mechanical and chemical stimuli.

As is known, the specific conditions of some industries cause the occurrence of occupational diseases. In a number of industries, teeth are damaged and frequently worn out. For workers involved in organic production and especially inorganic acids, upon examination, more or less uniform abrasion of all groups of teeth is revealed, there are no sharp edges. In some places exposed dense smooth dentin is visible. In persons with long experience Working in acid production plants wears teeth down to the neck. One of the first signs of abrasion of enamel under the influence of acid is the appearance of a feeling of soreness and roughness of the surface of the teeth. The change from a feeling of soreness to pain indicates the progression of the process. Chewing conditions may change. Upon examination, a loss of the natural color of the tooth enamel is revealed, which is especially visible when dried; a slight waviness of the enamel surface may be observed.

Individuals working in enterprises where there are excess mechanical particles in the air also experience increased tooth wear.

Often, increased tooth wear occurs in a number of endocrine disorders - dysfunction of the thyroid, parathyroid glands, pituitary gland, etc. The mechanism of erasure in this case is due to a decrease in the structural resistance of tissues. In particular, increased abrasion is observed with fluorosis, marble disease, Stainton-Candepont syndrome, primary underdevelopment of enamel and dentin.

For therapeutic dentistry, according to M.I. Groshikova, the most convenient clinical-anatomical classification is based on localization and degree of erasure.

Grade I- slight abrasion of the enamel of the cusps and cutting edges of the crowns of the teeth.

Degree II - abrasion of the enamel of the cusps of the fangs, small and large molars and the cutting edges of the incisors with exposure of the surface layers of dentin.

Grade III- abrasion of enamel and a significant part of dentin to the level of the coronal cavity of the tooth.

Abroad, the Bracco classification is most widespread. He distinguishes 4 degrees of abrasion: the first is characterized by abrasion of the enamel of the cutting edges and tubercles, the second - complete abrasion of the tubercles with exposure of dentin to 1/3 of the height of the crown, the third - a further decrease in the height of the crowns with the disappearance of the entire middle third of the crown, the fourth - the spread of the process to the level of the neck tooth

Beginners clinical manifestation The abrasion of teeth is caused by their increased sensitivity to temperature stimuli. As the process deepens, pain from chemical irritants, and then mechanical ones, may occur.

In most patients, despite pronounced degrees of abrasion, the sensitivity of the pulp remains within normal limits or is slightly reduced. Thus, in 58% of patients with tooth wear, the pulp response to electric current turned out to be normal, in 42% it was reduced to various levels (ranging from 7 to 100 μA or more). Most often, the decrease in electrical excitability of teeth ranged from 6 to 20 μA.

Pathological picture. Pathological changes depend on the degree of erasure. At initial manifestations When only slight abrasion occurs on the cusps and along the cutting edge, a more intense deposition of replacement dentin is noted according to the area of ​​abrasion. With more pronounced abrasion, along with significant deposition of replacement dentin, obstruction of the dentinal tubules is observed. Pronounced changes occur in the pulp: a decrease in the number of odontoblasts, their vacuolization, and reticular atrophy. Petrification is observed in the central layers of the pulp, especially in the root pulp.

At III degree After abrasion, pronounced sclerosis of dentin is observed, the tooth cavity in the coronal part is almost completely filled with replacement dentin, the pulp is atrophic. The number of odontoblasts is significantly reduced, and degenerative processes occur in them. The channels are difficult to navigate.

Treatment. The degree of abrasion of hard dental tissues largely determines the treatment. Thus, with I and II degrees of erasure, the main goal of treatment is to stabilize the process, prevent further progression erasing. For this purpose, inlays (preferably from alloys) can be made for antagonist teeth, mainly large molars. long time not susceptible to abrasion. It is possible to produce metal crowns(preferably made of alloys). If erasure is due to deletion significant amount teeth, then it is necessary to restore the dentition with a prosthesis (removable or non-removable according to indications).

Often, the abrasion of tooth tissue is accompanied by hyperesthesia, which requires appropriate treatment (see. Hyperesthesia of hard dental tissues).

Significant treatment difficulties arise with grade III abrasion, accompanied by a pronounced decrease in bite height. In such cases, the previous bite height is restored with fixed or removable dentures. Direct indications for this are complaints of pain in the area of ​​the temporomandibular joints, burning and pain in the tongue, which is a consequence of changes in the position of the articular head in the articular fossa.

Figure 5.12. Wedge-shaped tooth defect, a - diagram; b - appearance.

Treatment is usually orthopedic, sometimes long-term, with intermediate production of medical devices. The main goal is to create a position of the dentition that would ensure the physiological position of the articular head in the articular fossa. It is important that this jaw position be maintained in the future.

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3.3.2. Histological structure, chemical composition and functions of hard tissues of the tooth Enamel (enamelum). This tissue, covering the crown of the tooth, is the hardest in the body (250–800 Vickers units). On the chewing surface its thickness is 1.5–1.7 mm; on the lateral surfaces it is significantly

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5.2.5. Necrosis of hard dental tissues Clinical picture. The manifestation of necrosis begins with the loss of shine of the enamel and the appearance of chalky spots, which then become dark brown. In the center of the lesion, softening and formation of a defect are observed. At the same time, the enamel

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6.6.1. Principles and techniques of preparation of hard dental tissues for caries Cavity preparation is important stage treatment of dental caries, since only its correct implementation eliminates further destruction of hard tissues and ensures reliable fixation

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Self-persuasions aimed at developing strong ideological positions in patients regarding treatment, helping them avoid a breakdown (dieting disorder) “I made a firm decision to lose weight, restore my health, because I have a strong character, strong will. Nothing is not

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Abrasion of tooth tissue occurs in every person, which is the result of the physiological function of chewing.

Tooth wear can be physiological or pathological.

Physiological abrasion of teeth. Depending on age, the degree of physiological tooth wear increases. Normally, by the age of 40, the enamel of the tubercles of the chewing teeth wears off, and by the age of 50-60, abrasion is manifested by a significant loss of enamel on the tubercles of the chewing teeth and shortening of the crowns of the incisors.

Pathological abrasion of teeth. This term refers to early, at a young age, and highly pronounced loss of hard tissue in one tooth, in a group or in all teeth.

Etiology and pathogenesis. The causes of pathological abrasion of teeth can be the following factors: the state of the bite (for example, with a straight bite, the chewing surface of the lateral and cutting edges of the front teeth are subject to abrasion), overload due to loss of teeth, improper design of dentures, household and professional harmful influences, as well as the formation of defective tissue structures .

Classification. For therapeutic dentistry, the most convenient clinical-anatomical classification is based on the location and degree of abrasion (M. Groshikov, 1985), according to which three degrees of pathological abrasion are distinguished.

Grade I - slight abrasion of the enamel of the cusps and cutting edges of the crowns of the teeth.

Grade II- abrasion of the enamel of the canine tubercles, small and large molars and cutting edges of the incisors with exposure of the surface layers of dentin.

Grade III- abrasion of enamel and a significant part of dentin to the level of the coronal cavity of the tooth

Clinical picture. Depending on the degree of pathological abrasion, patients may complain of increased tooth sensitivity from temperature, mechanical and chemical irritants. It is also possible that there will be no complaints, since as the enamel and dentin wear away, replacement dentin is deposited due to the plastic function of the pulp. More often, patients complain of injury to the soft tissues of the oral cavity by the sharp edges of the enamel, which are formed due to more intense abrasion of dentin compared to enamel.

As the cusps of the chewing surface wear away with age, the abrasion of the incisors progresses intensively. The length of the incisor crowns decreases and by the age of 35-40 it decreases by 1/3-½. In this case, instead of a cutting edge, significant areas are formed on the incisors, in the center of which dentin is visible. If treatment is not carried out, tissue abrasion progresses rapidly and the crowns of the teeth become significantly shorter. In such cases, there are signs of a decrease in the lower third of the face, which is manifested in the formation of folds at the corners of the mouth. In persons with a significant decrease in bite, changes in the temporomandibular joint may occur and, as a result, burning or pain in the oral mucosa, hearing loss and other symptoms characteristic of low bite syndrome may occur.

In most patients with pathological abrasion, the EDI ranges from 6 to 20 μA.

Pathological picture. Pathological changes depend on the degree of erasure.

Grade I - Accordingly, more intense deposition of replacement dentin is observed in the area of ​​abrasion.

Grade II- along with significant deposition of replacement dentin, obturation of dentinal tubules is observed. Pronounced changes occur in the pulp: a decrease in the number of odontoblasts, their vacuolization. Petrification is observed in the central layers of the pulp, especially in the root pulp.

Grade III - pronounced sclerosis of dentin, the tooth cavity in the coronal part is almost completely filled with replacement dentin, the pulp is atrophic. The channels are difficult to navigate.

Treatment. The degree of abrasion of hard dental tissues largely determines the treatment. Thus, with degrees I and II of abrasion, the main goal of treatment is to stabilize the process and prevent further progression of abrasion. For this purpose, inlays (preferably from alloys) that are resistant to abrasion for a long time can be made on antagonist teeth, mainly large molars. You can also make metal crowns (preferably from alloys). If the abrasion is caused by the removal of a significant number of teeth, then it is necessary to restore the dentition with a prosthesis (removable or fixed according to indications).

Often, the abrasion of tooth tissue is accompanied by hyperesthesia, which requires appropriate treatment (see. Hyperesthesia of hard dental tissues).

Significant treatment difficulties arise with grade III abrasion, accompanied by a pronounced decrease in bite height. In such cases, the previous bite height is restored with fixed or removable dentures. Direct indications for this are complaints of pain in the area of ​​the temporomandibular joints, burning and pain in the tongue, which is a consequence of changes in the position of the articular head in the articular fossa. Treatment is usually orthopedic, sometimes long-term, with intermediate production of medical devices. The main goal is to create a position of the dentition that would ensure the physiological position of the articular head in the articular fossa. It is important that this jaw position be maintained in the future.

Wedge-shaped defect

The name of this pathological change is due to the shape of the defect in the hard tissues of the tooth (wedge type). The wedge-shaped defect is localized at the necks of the teeth of the upper and mandible, on the vestibular surfaces.

This type of non-carious lesion of hard tooth tissues is more common in middle-aged and elderly people and is often combined with periodontal disease.

Wedge-shaped defects most often affect canines and premolars, and the lesions can be single, but more often they are multiple, located on symmetrical teeth.

Etiology and pathogenesis. In the etiology of a wedge-shaped defect, mechanical and chemical theories occupy an increasingly important place. According to the first, a wedge-shaped defect occurs under the influence of mechanical factors. In particular, it is believed that the defect is formed as a result of exposure to a toothbrush. This is confirmed by the fact that it is most pronounced on the canines and premolars - teeth protruding from the dentition. Clinical observations have established that in individuals who have more developed right hand(right-handed), the defects on the left are more pronounced, since they brush the teeth of the left side more intensively. Left-handers who brush their teeth more intensively right side, defects are more pronounced on the right.

An objection to the mechanical theory is evidence that a wedge-shaped defect also occurs in animals and in people who do not brush their teeth at all.

Statements that acids play an important role in the occurrence of a wedge-shaped defect are unconvincing, since in other areas, including the cervical region of the interdental spaces, defects do not arise. However, acids entering the oral cavity can contribute to the rapid progression of already occurring abrasion of the tooth tissue at the neck.

Clinical picture. In most cases, a wedge-shaped defect is not accompanied by painful sensations. Sometimes patients only point out a tissue defect at the neck of the tooth. Usually it progresses slowly, and as it deepens, the contour does not change and disintegration and softening do not occur. In rare cases, quickly passing pain appears from temperature, chemical and mechanical stimuli. The quiet course or appearance of pain depends on the speed of loss of hard tissue. With slow abrasion, when replacement dentin is intensively deposited, pain does not occur. In cases where replacement dentin is deposited more slowly than tissue abrasion occurs, pain occurs.

The defect is formed by the coronal plane, which is located horizontally, and the second plane - the gingival plane, located at an acute angle. The walls of the defect are dense, shiny, and smooth. In cases where the defect comes close to the tooth cavity, its contours are visible. However, the tooth cavity is never opened. The wedge-shaped defect can reach such a depth that under the influence of mechanical load the tooth crown can break off. In most cases, probing is painless.

A wedge-shaped defect is often accompanied by gum recession.

Pathanatomy. There is a decrease in interprismatic spaces, obturation of dentinal tubules, and atrophy phenomena in the pulp.

Differential diagnosis. A wedge-shaped defect is differentiated from diseases of non-carious origin: erosion of hard dental tissues, cervical enamel necrosis, superficial and intermediate dental caries (see table).

Treatment. At the initial manifestations of a defect, measures are taken to stabilize the process. For this purpose, drugs are used that increase the resistance of hard dental tissues (applications of 10% calcium gluconate solution, 2% sodium fluoride solution, 75% fluoride paste). In addition, precautions are taken to reduce the mechanical impact on the teeth. Used for cleaning teeth soft brushes, use pastes containing fluoride or having a remineralizing effect. The movements of the toothbrush should be vertical and circular.

In the presence of pronounced defects filling of hard tissues is recommended. The most convenient filling materials are composite filling materials, which can be used to fill wedge-shaped defects without preparation. For deep defects, it is necessary to make artificial crowns.

Tooth erosion

Erosion is a defect in the hard tissues of the tooth localized on the vestibular surface and shaped like a saucer.

Etiology and pathogenesis have not been completely clarified. Some authors believe that tooth erosion, like a wedge-shaped defect, arises solely from the mechanical impact of a toothbrush. Others believe that erosion is caused by eating large quantity citrus fruits and their juices.

Yu. M. Maksimovsky (1981) important role in the pathogenesis of erosion of hard dental tissues is attributed to endocrine disorders and, in particular, hyperfunction of the thyroid gland. According to him, one of the symptoms of this disease is an increase in saliva secretion and a decrease in the viscosity of oral fluid, which cannot but affect the condition of the hard tissues of the tooth.

Localization. Erosion of hard dental tissues appears mainly on the symmetrical surfaces of the central and lateral incisors of the upper jaw, as well as on the canines and small molars of both jaws. Defects are located on the vestibular surfaces in the area of ​​the equators of the teeth. The lesion is symmetrical. There are practically no erosions on large molars and on the incisors of the lower jaw.

Classification. There are two clinical stages of erosion - active and stabilized, although in general any erosion of enamel and dentin is characterized by a chronic course.

For active stage A rapidly progressing loss of hard dental tissue is typical, which is accompanied by increased sensitivity of the affected area to various kinds of external stimuli (the phenomenon of hyperesthesia).

Stabilized stage erosion is characterized by a slower and calmer flow. Another sign is the absence of tissue hyperesthesia.

There are three degrees of erosion based on the depth of the lesion:

grade I, or initial, - damage to only the superficial layers of enamel;

degree II, or average,- damage to the entire thickness of the enamel up to the enamel-dentin junction;

grade III, or deep, - when the surface layers of dentin are also affected.

Clinical picture. Erosion is an oval or rounded enamel defect located on the most convex part of the vestibular surface of the tooth crown. The bottom of the erosion is smooth, shiny and hard.



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